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. Author manuscript; available in PMC: 2019 Feb 27.
Published in final edited form as: J Environ Pathol Toxicol Oncol. 2018;37(4):317–329. doi: 10.1615/JEnvironPatholToxicolOncol.2018028689

FIG. 1:

FIG. 1:

C(VI)-transformed bronchial airway epithelial cells (BEAS-2B) do not have mitochondrial respiratory dysfunction. (A) Mitochondrial respiration profile for BEAS-2B cells with the relevant injection strategy for the Seahorse Analyzer mitochondrial stress test. (B) Oxygen consumption data for BEAS-2B and B2B-CrT cells presented as a baseline percentage to the third oxygen consumption read. (C) Basal respiration, maximal respiration, and spare respiratory capacity for the BEAS-2B and B2B-CrT cells. (D) Proton leak, non-mitochondrial oxygen consumption, and coupled respiration for BEAS-2B and B2B-CrT cells. (E) Mitochondrial coupling efficiency for BEAS-2B and B2B-CrT cells. Data are the average of at least three experiments ± SEM. *p < 0.05.