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. 2019 Feb 27;9:2952. doi: 10.1038/s41598-019-39367-z

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Functional dysconnectivity of the PFC, motor, and somatosensory thalamus seeds in schizophrenia. For the thalamic PFC (A) and motor (B) seeds, patients with schizophrenia showed reduced functional connectivity with the dorsolateral PFC, dorsal anterior cingulate cortex, inferior parietal lobule, and cerebellum (warm colors), and increased functional connectivity with the pre- and postcentral gyri, superior and middle temporal gyri, lateral and medial occipital regions (cold colors), relative to healthy subjects. In contrast, functional connectivity of the thalamus somatosensory seed (C) was mainly restricted to the postcentral gyrus and lateral occipital region.