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. 2019 Feb 27;10:971. doi: 10.1038/s41467-019-08665-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — ICA component correlated with the thalamus in P12 (“SEEG-triggered analysis”). a In black, ICA time course and in color (red), time course on the SEEG contact where significant correlation with ICA was measured. b MEG topography of ICA component. c 3D representation of MEG-SEEG correlation. Color (from yellow to red, refer to color bar) and sphere dimension correspond to the correlation value. d MRI (T1) with reconstructed SEEG electrodes (showing electrode TH’); the arrow indicates the contact with the highest correlation value, located in the left thalamus. e Source localization (single dipole) of the ICA component overlaid on patient MRI, which includes regions sampled by mesial contacts of electrode TH’