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. 2018 Oct 12;42(1):64–80. doi: 10.1007/s10753-018-0873-9

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© The Author(s) 2018

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 8 — Catalpol inhibited HCY-induced ER stress in HAECs. (a) Effect of catalpol on GRP78 mRNA expression level induced by HCY. (b) Effect of catalpol on ATF4 mRNA expression level induced by HCY. (c) Effect of catalpol on GRP78 protein expression level induced by HCY. (d) Effect of catalpol on ATF4 protein expression level induced by HCY. (e) Effect of catalpol on P-PERK protein expression level induced by HCY. (f) Effect of catalpol on P-eIF2α protein expression level induced by HCY. (g) Effect of catalpol on P-IRE1α protein expression level induced by HCY. (h) Effect of catalpol on ATF6 protein expression level induced by HCY. Results represent the mean ± S.D. from three independent experiments. ^#p < 0.05 vs. control, ^*p < 0.05 vs. HCY, ^##p < 0.01 vs. control, ^**p < 0.01 vs. HCY.