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. 2019 Feb 25;10:240. doi: 10.3389/fimmu.2019.00240

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Copyright © 2019 Trojnar, Józsi, Szabó, Réti, Farkas, Kelen, Reusz, Szabó, Garam, Mikes, Sinkovits, Mező, Csuka and Prohászka.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effect of PTX3 on AP activity in vitro. The effect of recombinant human PTX3 on AP hemolytic activity (A) and AP-mediated C9 deposition (B) is shown in percent compared to buffer control added to pooled serum of healthy individuals. Data represent mean of 3 times repeated experiments with technical duplicates, error bars indicate the standard error of mean. Statistical analysis was performed with the Kruskal-Wallis test corrected for multiple comparisons using the Dunn's post hoc test. Statistical significance is indicated by asterisks (*p < 0.05; ***p < 0.001). AP, alternative pathway; PTX3, pentraxin-3.