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. 2019 Jan 21;10(3):492–500. doi: 10.1111/1759-7714.12962

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© 2019 The Authors. Thoracic Cancer published by China Lung Oncology Group and John Wiley & Sons Australia, Ltd

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.

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Scutellarin (SCU) suppresses proliferation in non‐small cell lung cancer (NSCLC) A549 cells. (a) The chemical structure of SCU. (b) A549 cells were treated with SCU (0, 200, 400, 600 μM) for 12, 24 and 48 hours. Cell viability was detected by Cell Counting Kit‐8 assay. (c) The proliferative effect of SCU on A549 cells detected by colony formation assay. Data are expressed as means ± standard deviation from three independent experiments. ^* P < 0.05, ^** P < 0.01 vs. the control group. () 12 hours, () 24 hours and () 48 hours.

Inline graphic — Scutellarin (SCU) suppresses proliferation in non‐small cell lung cancer (NSCLC) A549 cells. (a) The chemical structure of SCU. (b) A549 cells were treated with SCU (0, 200, 400, 600 μM) for 12, 24 and 48 hours. Cell viability was detected by Cell Counting Kit‐8 assay. (c) The proliferative effect of SCU on A549 cells detected by colony formation assay. Data are expressed as means ± standard deviation from three independent experiments. ^* P < 0.05, ^** P < 0.01 vs. the control group. () 12 hours, () 24 hours and () 48 hours.