Table 2.
General characteristics of the 151 included neonates
| Sexa (n = 125) |
Male 37.7% |
Female 45.0% |
Missing 17.2% |
| Gestational agea (n = 114) |
Term 62.9% |
Preterm 12.6% |
Missing 24.5% |
| N (%) | |||
| Etiology (n = 151) | |||
| Hypoxic‐ischemic encephalopathy | 6 (4.0) | ||
| Cortical malformations | 3 (2.0) | ||
| CNS infections | 4 (2.6) | ||
| Metabolic disorders | |||
| Electrolyte imbalance | 3 (2.0) | ||
| Inborn errors of metabolism | 3 (2.0) | ||
| Vitamin‐related disorders | 11 (7.3) | ||
| Withdrawal seizures | 2 (1.3) | ||
| Genetics | |||
| Channelopathies | 67 (44.4) | ||
| Chromosomal disorders | 3 (2.0) | ||
| Other gene disorders | 7 (4.6) | ||
| Vascular | |||
| Stroke | 25 (16.6) | ||
| Hemorrhage | 8 (5.3) | ||
| Undetermined/unknown | 9 (6.0) | ||
| Seizure type (n = 151) | |||
| Sequential | 62 (41.1) | ||
| Clonic | 36 (23.8) | ||
| Tonic | 26 (17.2) | ||
| Autonomic | 14 (9.3) | ||
| Myoclonic | 9 (6.0) | ||
| Spasms | 3 (2.0) | ||
| Automatisms | 1 (0.7) | ||
| EEG (n = 151)b | |||
| Focal | 56 (37.1) | ||
| Burst‐suppression | 48 (31.8) | ||
| Multifocal | 46 (30.5) | ||
| Generalized | 1 (0.7) | ||
a
Information not available for all newborns.
b
Information related to ictal EEG except in some cases of burst‐suppression (BS). Burst‐suppression was described as an ictal pattern in 2 neonates and as an interictal in 8; in the remining cases, it was not clearly defined as an ictal or interictal pattern/background abnormality.