(A) Cohorts, (B) Somatic mutational loads, (C) Ploidy and cellularity, (D) Waddell class, (E) Genomic complexity (the proportion of the tumor’s genome with copy number deviating from its ploidy), (F) Mutational signatures, (G) Driver gene alterations, (H) Copy number variation, (I) Cell cycle progression, (J) Hypoxia expression, (K) RNA subtypes. See also Figure S1.