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. 2019 Mar 5;9:3535. doi: 10.1038/s41598-019-40419-7

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The protein content of adiponectin in the EAT. (a) Representative images of immunohistochemical staining of adiponectin in the EAT of the non-CAD and CAD patients. Adiponectin is stained in dark brown in the cytosol around the lipid droplet in the adipocyte of the EAT (see red arrows), and intensity of adiponectin staining appears to be weak in a CAD patient compared to a non-CAD patient. (b) The protein levels of adiponectin in EAT in non-CAD (n = 11) and CAD patients (n = 13). Bar: mean ± SD. *P < 0.05. (c–g) The association the protein content of adiponectin and the mitochondrial respiratory capacity in the EAT. (h) The association between the protein content of adiponectin and the severity of coronary artery stenosis. White and black circles indicate non-CAD (n = 11) and CAD patients (n = 13), respectively. A solid line indicates a significant correlation in all patients and a dashed line indicates a significant correlation only in CAD patients. Abbreviations are explained in the Fig. 1 legend.