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. 2019 Mar 5;16:17. doi: 10.1186/s12986-019-0344-9

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Protocols of the 4-week exercise and treatment with PPARγ agonist and antagonist. The rats in Con, OB and DM groups kept sedentary life while EOB, EDM, EDG and EDP rats participated in a moderate-intensity aerobic exercise on a treadmill with gradually increasing intensity and duration, once a day and 6 days/week, and at 30 min before exercise the rats in EDP and EDG groups were intragastrically administered PPARγ agonist pioglitazone (10 mg/kg body weight) and PPARγ antagonist GW9662 (1 mg/kg body weight) respectively. Con: control; OB: obesity; DM: diabetes mellitus; EOB: exercised OB; EDM: exercised DM. EDP: EDM + pioglitazone; EDG: EDM + GW9662