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. Author manuscript; available in PMC: 2019 Dec 1.
Published in final edited form as: J Mol Cell Cardiol. 2018 Oct 22;125:87–97. doi: 10.1016/j.yjmcc.2018.10.011

Fig 1.

Fig 1.

Angiotensin II (Ang II) or phenylephrine (PE) induces calmodulin (CaM) movement from RyR2 to nucleus. (A) Immunofluorescent images of adult cardiomyocytes during control, 30min exposure to 10 μmol/L Ang II, or 100 μmol/L PE. RyR2 (red) and CaM (green) are shown. (B) Summary of CaM translocation treatment with Ang II and PE corresponding to the experiment in panel A. (C) Kinetics of CaM movements of RyR2 binding CaM and nucleus CaM treated with 10 μmol/L Ang II. Data are mean ± SE, n values on bars, *P<0.05 vs. Control (Ctl) (one-way ANOVA followed by tukey-kramer’s post-test).