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. 2019 Mar 11;9:4079. doi: 10.1038/s41598-019-40606-6

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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The effects of VML and voluntary wheel running on muscle strength and oxidative capacity 4 weeks post injury. (a) Mitochondrial respiratory function normalized by grams wet weight of permeabilized muscle fibers (n ≥ 12 permeabilized fiber bundles from n = 6 mice for each condition) did not adapt to exercise training in the VML injured limb. (b) Mitochondrial respiratory function normalized to citrate synthase enzyme activity (see Table 1). (c) Wheel running distance of control and VML injured mice. Peak isometric torque normalized to (d) body mass and (e) plantarflexor mass in the VML injured limb was significantly greater after voluntary wheel running. Data analyzed by one-way ANOVA, *P < 0.05. Error bars represent means ± SD.