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. 2019 Feb 20;43(4):1806–1816. doi: 10.3892/ijmm.2019.4102

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Copyright: © Cheng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Activation of TGF-β reduces the effects of miRNA-30e on the oxidative stress in vitro model. (A) TGF-β, (B) Smad2 and (C) Nox4 protein expression by statistical analysis and (D) western blotting analysis, (E) MDA, (F) SOD, (G) GSH and (H) GSH-PX (I) ROS levels and (J) green fluorescent protein. ^**P<0.01 vs. the negative normal group, ^##P<0.01 vs. the over-expression of microRNA-30e group. miR/miRNA, microRNA; TGF, transforming growth factor; Nox4, NAPDH oxidase 4; Smad, mothers against decapentaplegic homolog; Negative, negative control group; miR-30e, over-expression of microRNA-30e group; TGF-β, over-expression of microRNA-30e and TGF-β plasmid group.

Activation of TGF-β reduces the effects of miRNA-30e on the oxidative stress in vitro model. (A) TGF-β, (B) Smad2 and (C) Nox4 protein expression by statistical analysis and (D) western blotting analysis, (E) MDA, (F) SOD, (G) GSH and (H) GSH-PX (I) ROS levels and (J) green fluorescent protein. ^**P<0.01 vs. the negative normal group, ^##P<0.01 vs. the over-expression of microRNA-30e group. miR/miRNA, microRNA; TGF, transforming growth factor; Nox4, NAPDH oxidase 4; Smad, mothers against decapentaplegic homolog; Negative, negative control group; miR-30e, over-expression of microRNA-30e group; TGF-β, over-expression of microRNA-30e and TGF-β plasmid group.