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. 2019 Mar 13;9:4388. doi: 10.1038/s41598-019-40934-7

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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CA improved glucose consumption and glycogen stimulate led by cAMP and DEX induced T2D cell model. (A) Model building schematic diagram; (B) PEPCK mRNA expression in different cAMP and DEX treated group; After CA treatment, glucose consumption (C), glycogen stimulate (D) and PEPCK mRNA expression (E) were analyzed; (F) Antiproliferative effect of CA on HepG2 cells were analyzed by CCK8. * Indicates a significant difference from the control at P < 0.05, **P < 0.01, ***P < 0.0001. C, cAMP; D, DEX; M, metformin.