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. 2019 Mar 7;9:57. doi: 10.3389/fcimb.2019.00057

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Copyright © 2019 Yang, Yue and Xiong.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Dpep2 repressed pro-inflammatory responses in macrophages by modulating NF-κB signaling. (A) BMDMs derived from Dpep2^fl/fl LyzM-Cre⁺ or Dpep2^fl/fl mice were treated with CVB3 (MOI = 10) for 15, 30, 60, 120, or 240 min, and the levels of p65, IKKα, IκB, p-p65, p-IKKα, and p-IκB were analyzed by Western blot. (B) Band intensities were quantified by ImageJ software and normalized to GAPDH. Data were from one representative experiment among two independent experiments with similar results and were presented as the means ± S.E.M. ^**p < 0.01; ^***p < 0.001; ns, no significance.