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. 2019 Mar 8;13:201. doi: 10.3389/fnins.2019.00201

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Copyright © 2019 Hass and Barnstable.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Increased Ucp2 expression in RGCs but not astrocytes and müller glia decreases oxidative stress. (A) Representative labeling of fixed retinas from bead-injected Ucp2^KI (n = 7), Ucp2^KI; Gfap-creER^T2 (n = 3), and Ucp2^KI; Thy1-creER^T2 (n = 4) eyes for oxidative stress-derived protein carbonylation. A single section was labeled without using the 2′,4′-dinitrophenylhydrazine derivitization reagent to confirm the specificity of the primary antibody to toward dinitrophenylhydrazones. (B) The intensity of DAB labeling is quantified. ^∗p < 0.05.