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. 2019 Mar 15;11:50. doi: 10.1186/s13148-019-0643-z

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Differences in DLK1-MEG3 CpG site methylation and imprinted gene expression based in AML MNCs based on patient risk group and FLT3 mutation status. Kruskal-Wallis (a) or Mann-Whitney (c) analysis of methylation values for particular CpG site amplicons depending on AML patients’ risk groups (a) or presence of FLT3 mutation (c). WT, wild-type; Mut, mutated. Kruskal-Wallis (b) or Mann-Whitney (d) analysis of DLK1 and MEG3 and the DLK1/MEG3 expression ratio in MNCs from AML patients based on their risk group (b) or presence of FLT3 mutation (d). WT, wild-type; Mut, mutated