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. 2019 Jan 30;19(4):2581–2590. doi: 10.3892/mmr.2019.9908

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Copyright: © Qin et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — RP suppresses the MI/R-induced apoptosis of cardiomyocytes. Flow cytometry was performed to assess the cell apoptosis of cardiomyocytes, treated with MI/R, 20 µM RP+MI/R, 40 µM RP+MI/R and 80 µM RP+MI/R. ^#P<0.05 vs. Control; *P<0.05 vs. MI/R. Rp, rhynchophylline; MI/R, myocardial ischemia-reperfusion; PI, propidium iodide; FITC, fluorescein isothiocyanate.