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. 2019 Mar;17(3):232–246. doi: 10.2174/1570159X16666180828100002

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© 2019 Bentham Science Publishers

This is an open access article licensed under the terms of the Creative Commons Attribution-Non-Commercial 4.0 International Public License (CC BY-NC 4.0) (https://creativecommons.org/licenses/by-nc/4.0/legalcode), which permits unrestricted, non-commercial use, distribution and reproduction in any medium, provided the work is properly cited.

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Fig. (2) — Mechanisms of peroxisome proliferator-activated receptor gamma (PPARγ) transrepression- (A) Co repressor interference mechanism- co-repressor complexes occupied on the promoters region of NF-kB is stabilize by PPARγ agonist by SUMOylation of PPARγ ligand binding domain within PPARγ and inhibit proinflammatory inhibitors (B) Cross coupling- direct binding of PPARγ to NF-kB forming inactive transcriptional complexes and inhibit proinflammatory inhibitors. (The color version of the figure is available in the electronic copy of the article).