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. 2019 Feb 12;17(4):2465–2472. doi: 10.3892/etm.2019.7256

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Copyright: © Chen et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — miR-125a upregulation suppressed PI3K/Akt/mTOR signalling pathway activation in an in vitro model of thyroiditis. (A) The relative quantification of (A) PI3K (B) p-Akt and (C) p-mTOR protein expressions was calculated from western blot analysis results. (D) Western blot analysis of PI3K, p-Akt and p-mTOR expression. Data are expressed as the mean ± standard deviation of independent experiments (n=3). ^##P<0.01 vs. control group. miR-125a, microRNA-125a; PI3K, phosphoinositide 3-kinase; Akt, protein kinase B; mTOR, mechanistic target of rapamycin; p, phosphorylated.