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. 2019 Mar 21;26:26. doi: 10.1186/s12929-019-0514-0

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — The CRHR2 mRNA levels and protein expressions in the mPFC, hippocampus, and amygdala. OXT could decrease SPS-increased mPFC CRHR2 mRNA level, and this effect could be blocked by ASB (a). OXT and ASB reduced the hippocampus CRHR2 mRNA level in SPS rats (b). SPS and drugs (OXT and ASB) did not affect the CRHR2 mRNA level in amygdala (C). SPS and drugs (OXT and ASB) did not affec the CRHR2 protein expressions in the mPFC, hippocampus, and amygdala, excepting ASB reduced hippocampus CRHR2 expression (D-F). SPS = single prolonged stress, VEH = vehicle, OXT = oxytocin, ASB = atosiban, OXTR = oxytocin receptor. Bars represent mean ± SEM; *p < 0.05, **p < 0.01, ***p < 0.01, compared with Control-Veh; ##p < 0.01, ###p < 0.001, compared with Control-OXT. N = 5 for the mRNA levels (A-C); N = 4 for the protein expressions (d-f)