Figure 1.

Schematic representation of the cellular functions of OGE regulators in plant response to salinity. Salt stress can perturb chloroplast or mitochondrion homeostasis and this would be communicated to the nucleus through retrograde signals, leading to changes in nuclear gene expression. In turn, this would activate the expression of OGE regulators (e.g., mTERFs, PPRs or DEAD-box RHs proteins), which would result in anterograde signalling responses to adjust organellar function to salinity. In the figure, inside a mitochondrion (purple) and chloroplast (green), only those molecularly-characterized OGE regulators whose mutations lead to altered responses to salinity are depicted. The OGE processes affected in these mutants, as well as their enhanced (↑) or reduced (↓) sensitivity to salt stress, are shown (see also Table 1 for further information).