Table 1.
Patients (n = 35) | |
---|---|
Age | 58 ± 9 years |
Male | 29 (83%) |
Hypertension | 11 (26%) |
Hypercholesterolemia | 7 (17%) |
Total cholesterol, mmol/L | 4.9±0.8 mmol/L |
Diabetes | 1 (2%) |
Smoking | |
Non | 10 (24%) |
Active | 18 (43%) |
Ex | 14 (33%) |
Family history of premature CAD | 17 (41%) |
Peripheral arterial disease | 0 |
Infarct location | |
LAD | 17 (49%) |
RCA | 15 (42%) |
LCX | 2 (6%) |
LM | 1 (3%) |
TIMI flow prior to pPCI | |
0 | 17 (49%) |
1 | 8 (23%) |
2 | 5 (14%) |
3 | 5 (14%) |
TIMI flow post-pPCI | |
0 | 1 (2%) |
1 | 0 |
2 | 3 (9%) |
3 | 31 (89%) |
Peak troponin T, ng/mL | 5769 ± 6201 |
Peak CK-MB, U/I | 247 ± 175 |
Ejection fraction post-pPCI at discharge, % | 43.8 ± 9.6 |
Time from symptom-onset to PCI, minutes (IQR) | 165 (120–273) |
Medication during follow-up | |
ACE/ARB | 11 (31%) |
Beta-blockers | 31 (88%) |
Statins | 35 (100%) |
Thiazide/loop diuretic | 6 (17%) |
Acetylsalicylic acid | 35 (100%) |
ADP antagonist | 35 (100%) |
CAD, coronary artery disease; LAD, left anterior ascending artery; RCA, right coronary artery; LCX, left circumflex artery; LM, left main artery; TIMI, thrombolysis in myocardial infarction; pPCI, post-primary percutaneous intervention; CK-MB, Creatine-kinase MB; IQR, interquartile range; ACE/ARB, angiotensin-converting-enzyme inhibitor/angiotensin receptor II blocker; ADP antagonist, adenosine diphosphate (ADP) receptor inhibitor