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. 2019 Feb 28;23(Suppl 1):S11–21. doi: 10.5213/inj.1938040.020

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Copyright © 2019 Korean Continence Society

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 3. — Mitochondrial and cytosolic cytochorome c, and anti- or pro-apoptotic protein in ischemia. (A) Cytochrome c abundance in mitochondrial (left) and cytosolic fraction (right) from hippocampal CA1 at 24 hours after ischemia. (B) Antiapoptotic protein, Bcl2. (C) Pro-apoptotic protein, Bax expression levels in whole cell lysate form hippocampal CA1 at 24 hours after ischemia. Quantifications were normalized by Cox IV for mitochondrial fraction and β-actin for cytosolic fraction and whole cell lysate expression (n=4 for each group). *P<0.05. ***P<0.001.