The NTF attachment of ADGRG6 to its CTF suppresses receptor activity,
thereby maintaining Schwann cells in an immature state and allowing for radial
sorting of cells to axons. Increased accumulation and binding of its ligand
laminin-211 removes the NTF, thereby allowing for
Stachel-dependent activation of ADGRG6, association with
Gαs, accumulation of cyclic AMP (cAMP), and the initiation
of cAMP-dependent myelinogenic gene programs and the myelination of peripheral
axons. Adapted from [30].