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. 2019 Mar 25;9(1):2045894019837876. doi: 10.1177/2045894019837876

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© The Author(s) 2019

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Fig. 8. — (a) H&E staining of the pulmonary arteries from Patients 1–9. Most arteries show varying degrees of medial thickening except Patient 4, despite extensive emphysema. (b) Immunofluorescence study depicting the expression of cav-1 (green) and smooth muscle α-actin (red) in pulmonary arteries. The control artery (Con) and the arteries from patients with chronic lung display the presence of endothelial cav-1. (c) Representative immunofluorescence study showing the expression of vWF (yellow/green) and smooth muscle α-actin (red) as merged figures. There is no loss of vWF in the group of patients with the lung diseases we studied.