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. 2018 Jul 11;27(8):1249–1255. doi: 10.1177/0963689718784957

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© The Author(s) 2018

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Fig. 2. — Weak phosphorylation of Akt induced by SDF-1α through the CXCR7 receptor and little inhibitory effects on CXCR7-mediated Akt phosphorylation by HC4319 and DV-1. (A) Expression of CXCR7 on infected CHO cells analyzed by flow cytometry. (B) Effects of SDF-1α on the phosphorylation of Akt and Erk in CXCR7^high CHO cells detected by Western blotting. (C) Effects of SDF-1α on the phosphorylation of Akt in wild-type CHO cells detected by Western blotting. (D) Effects of HC4319 and DV-1 on SDF-1α-induced phosphorylation of Akt in CXCR7^high CHO cells assessed by Western blotting.