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. 2018 Sep 28;82(2):143–150. doi: 10.4046/trd.2018.0027

Table 2. The mutation patterns of the katG and inhA genes in 206 patients with a genotypic INH resistance.

Group INH mono-resistance MDR-TB Total p-value
No. of subjects 93 (45.1) 113 (54.9) 206 (100)
katG mutation alone 55 (59.1) 79 (69.9) 134 (65.0) 0.107
 Mutant probe (+) 50 (53.8) 77 (68.1) 127 (61.7) 0.035
 Wild-type band loss alone 5 (5.4) 2 (1.8) 7 (3.4) 0.248
inhA mutation alone (%) 37 (39.8) 29 (25.7) 66 (32.0) 0.031
 Mutant probe (+) 33 (35.5) 29 (25.7) 62 (30.1) 0.126
 Wild-type band loss alone 4 (4.3) 0 (0) 4 (1.9) 0.040
Both gene mutation 1 (1.1) 5 (4.4) 6 (2.9) 0.225
katG (MUT1), inhA (MUT1) 0 (0) 2 (1.8) 2 (1.0)
katG (MUT1), inhA (WT1 loss) 1 (1.1) 0 (0) 1 (0.5)
katG (MUT1), inhA (WT2 loss) 0 (0) 1 (0.9) 1 (0.5)
katG (WT loss), inhA (MUT1) 0 (0) 1 (0.9) 1 (0.5)
katG (WT loss), inhA (WT2 loss) 0 (0) 1 (0.9) 1 (0.5)

Values are presented as number (%).

INH: isoniazid; MDR-TB: multidrug-resistant tuberculosis; katG WT: S315 region; katG MUT1: S315T[AGC315ACC]; inhA WT1: positions −15 to −16; inhA WT2: positions −8; inhA MUT1: C15T.