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. 2019 Jan 7;9(2):335–350. doi: 10.1016/j.apsb.2019.01.003

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© 2019 Chinese Pharmaceutical Association and Institute of Materia Medica, Chinese Academy of Medical Sciences. Production and hosting by Elsevier B.V.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 4 — Protective effect of compounds 33, 21 and 1b on brain infarct after 72 h reperfusion. (A) TTC staining analysis of the infarcted brain regions. (B) Quantitative analysis of the infracted brain regions. The ratios of infarct area to whole brain areas in individual rats were calculated. (C) Quantitative analysis of neurological score. Cerebral infarction in Sham-operated (Sham) or MACO-reperfusion rats from a representative animal that received normal saline (NS), Veh (polymer:DMSO:distilled water at 50 mg:1 mL:3 mL), or 33-nanoparticles (0.2 mg/kg), 21-nanoparticle (0.2 mg/kg), and 1b-nanoparticles (0.2 mg/kg) intracerebroventricularly 2 h before MCAO. Data are expressed as the mean±SD (n=3). ^###P < 0.001 significantly different from Sham group, ^***P < 0.001, ^*P < 0.05 significantly different from control group.