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. 2019 Jan 7;9(2):335–350. doi: 10.1016/j.apsb.2019.01.003

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© 2019 Chinese Pharmaceutical Association and Institute of Materia Medica, Chinese Academy of Medical Sciences. Production and hosting by Elsevier B.V.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 5 — Therapeutic effect of compound 33 on cerebral ischemia-reperfusion injury. (A) TTC staining analysis of the infarcted brain regions. Quantitative analysis of the infracted brain regions (B) and neurological score (C). (D) and (E) The locomotion trace of the mice in the chamber for a period of 5 min. All animals were divided into seven groups: sham-operated group, NS group, Veh (Cremophor:PBS at 3:97) group. 33 (15 mg/kg, 3 h) + MCAO group, 33 (15 mg/kg, 6 h) + MCAO group, ED (15 mg/kg, 3 h) + MCAO group and ED (15 mg/kg, 6 h) + MCAO group. All rats received drug intraperitoneally 3 or 6 h after MCAO. Data are expressed as the mean±SD (n=3). ^###P<0.001, ^##P<0.01 significantly different from Sham group, ^***P < 0.001, ^**P<0.01, ^*P < 0.05 significantly different from control group.