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. 2019 Mar 22;10:506. doi: 10.3389/fimmu.2019.00506

Table 2.

Phenotypic characteristics of ERdj5−/− mice and other mouse SS models.

Inflammatory infiltrates in SG SG tissue damage Serum AutoAbs Salivary hypofunction Female predilection Cytokines in serum Cytokines in SGs Parenchymal inflammation Other phenotypes
Knock-out ERdj5 KO +
B-cells, T-cells
(2:1 ratio)
+ +
in females
anti-Ro anti-La
+ + Elevated
♀: IL-23, IL-18
♂: IL-17A
Elevated
♀: IL-23, IL-10
Kidney, occasional infiltrations in liver nd
Id3 KO +
IFN-γ and IL-4 producing lymphocytes
nd +
Anti-Ro, Anti-La, Autoreactivity to ductal cells
+ nd nd Occasional infiltrations in kidney and lung Lachrymal gland inflammation and hypofunction. Tumors in numerous organs. Late onset of SS-like phenotype. Skin lesions by over-scratching
IkB-ζ KO +
Anti-Ro, Anti-La
nd nd nd nd Interstitial pneumonia Lachrymal gland inflammation, apoptosis, and hypofunction. Periocular dermatitis. Conjunctivitis. Splenomegaly. Lymphadenopathy. Elevated serum IgGs
Transgenic-overexpression BAFF +
B-cells predominantly
+ - for Anti–Ro- for Anti-La + nd nd Late onset nephritis. Occasional lung, liver infiltrations SLE-like. SS-like with age. Reduction in T-regs. Germinal centers in SGs. Hyperactive B-cells
IL-12 in
SJL strain
+ nd +
ANAs, Anti-La
+ + nd nd Lung inflammation Lachrymal gland inflammation. Mild autoimmune thyroiditis. Affected osmoregulation of SGs
Chimeric SS PBMCs in NOD-scid IL-2rγ (null) +
CD4 predominantly
nd nd + nd Elevated IFN-γ, IL-10, IL-17, IL-2, IL-6, TNFα nd nd Type-I diabetes. Lachrymal gland inflammation
Spontaneous models MRL/lpr +
IFN-γ and IL-17 producing CD4+
nd +
Anti-Ro, Anti-La
– for SG+
for lachrymal
nd IFN γ, IL-1β, IL-6, TNFα nd SLE. Lachrymal gland inflammation. Early death.
Aggressive autoimmune lympho-proliferative disorder
NOD +
Dynamic CD4+ PD1+ FOXP3-, mainly Th1
+ +
Anti-Ro, Anti-La
+ +
SG in ♀
Lachr. in ♂
IFNs IL-1β, IL-2, TNFα, IFN-γ, IL-10, IL-17, IL-12(p40) Lung inflammatory infiltrations. Kidney antibody deposition IDDM. Thyroiditis-like. SLE. Myasthenia. SS phenotype affected by housing conditions. Weak association with HLA haplotypes
NOD Aec1. Aec2 +
Progressive T-cell and B-cell infiltrations
+ + + + Differentially expressed ± IFNs ± IFNs. Elevated cytokines nd Anti-M3R antibodies. Lachrymal gland hypofunction in females
NZB/NZW F1 Augmented by incomplete Freund's adjuvant IP injection (F.adj. inj.) nd ANAs and anti-Ro augmented by F.adj.IP Augmented by F.adj. and polyI:C injection + nd Augmented by polyI:C injection Kidney SLE-like phenotype. Non-specific inflammatory stimuli. Conjunctivitis
Immunization Ro60 in BALB/c + nd nd + nd nd nd nd Anti-Ro, Anti La in saliva. Late onset. Requires multiple immunizations
Alum in NZM2758 + nd + ANAs + nd nd Inflammasome components” nd P2X7 receptor-dependent phenotypes”

+, SS trait is reported in the relevant literature; –, SS trait reported negative in the relevant literature; nd, not determined in the relevant literature; italics, trait discussed as possibility but not shown in experimental data.