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. 2019 Mar 29;38:141. doi: 10.1186/s13046-019-1119-x

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — miR-96-5p upregulation is stronger in TP53 mutated HNSCC and predicts local recurrence in HNSCC patients. a Box plot showing expression level of miR-96-5p in the tumoral and normal tissues from IRE cohort. b Box plot of the expression level of miR-96-5p in the tumoral and normal tissues from TCGA cohort. c Kaplan–Meier (KM) analysis showing correlation between the expression level of miR-96-5p and recurrence-free survival in the HNSCC patients from IRE cohort. d-e Comparison of miR-96-5p expression between TP53 mutated and wild type tumors from matched samples IRE cohort (d) and from matched samples TCGA cohort (e). N: number of samples, FC: Fold change, p: p-value