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. 2019 Mar 26;13:278. doi: 10.3389/fnins.2019.00278

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Copyright © 2019 Al-Amin, Chowdury, Saifullah, Alam, Jain, Hossain, Alam, Kazi, Ahmad, Raish, Alqahtani and Reza.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Treatment of levocarnitine and aluminum chloride on nitric oxide (NO) (A–F); in the prefrontal cortex (PFC) (A), striatum (ST) (B), parietal cortex (PC) (C), hypothalamus (HT) (D), hippocampus (HP) (E) and cerebellum (CB) (F). The groups were control, AlCl3 (aluminum chloride), AST (astaxanthin), AST+AlCl3 (aluminum chloride and astaxanthin) and AlCl3+LC (aluminum chloride and levocarnitine) and LC (levocarnitine). Data presented as mean ± SEM. n = 6 per group except AST (n = 3); and AlCL3+AST (n = 3); ^∗∗∗p < 0.001, ^∗∗p < 0.01, ^∗p < 0.05.