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. 2019 Feb 26;17(5):4393–4400. doi: 10.3892/ol.2019.10077

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Copyright: © Pang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — Effects of cell division cycle-associated protein 4 KD on the regulation of triple negative breast cancer cell xenograft growth in vivo. (A) Curves of tumor xenograft growth over time in nude mice. (B) Tumor xenograft weight. Differences in tumor xenograft weights between the NC and KD groups (P<0.05). (C) In vivo small animal imaging. Tumor cell xenografts were measured using fluorescence total radiant efficiency on days 22 (P>0.05), 29 (P<0.05) and 36 (P>0.05). (D) Tumor xenograft size. The xenografts were dissected from the subcutaneous tissues of the mice, and size was compared between the NC and KD groups. The data were expressed as the means ± standard deviation. KD, knockdown; NC, negative control.

Figure 6. — Effects of cell division cycle-associated protein 4 KD on the regulation of triple negative breast cancer cell xenograft growth in vivo. (A) Curves of tumor xenograft growth over time in nude mice. (B) Tumor xenograft weight. Differences in tumor xenograft weights between the NC and KD groups (P<0.05). (C) In vivo small animal imaging. Tumor cell xenografts were measured using fluorescence total radiant efficiency on days 22 (P>0.05), 29 (P<0.05) and 36 (P>0.05). (D) Tumor xenograft size. The xenografts were dissected from the subcutaneous tissues of the mice, and size was compared between the NC and KD groups. The data were expressed as the means ± standard deviation. KD, knockdown; NC, negative control.