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. 2019 Apr 2;9:5515. doi: 10.1038/s41598-019-41941-4

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Wound healing behaviour. (A). Relative wound density at 30 hr for high-grade serous (HGS) cell lines compared to non-serous (NS) cell lines (median ± IQR). HGS median = 14.36 ± 19.03, NS median = 61.38 ± 34.9 Mann-Whitney U test, p < 0.001, U = 1795. (B). Relative wound density at 30 hr for each cell line (median ± IQR). (C). Relative wound density over 60 hours (mean ± SEM). Two-way repeated measures ANOVA identifies two populations of cell lines (p < 0.001). Group A cell lines (COV318, TOV21G, OVCAR8) show no significant difference between relative wound density at >75% of time points (p < 0.005). Group B cell lines (A2780, PEO1, PEO4, FUOV1) show no significant difference between relative wound density at all time points (p < 0.005). (HGS: blue, NS: yellow, Control: grey). Experiments were carried out in triplicate on 3 separate occasions for each cell line. ***p < 0.001.