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. 2019 Mar 15;36(7):1060–1079. doi: 10.1089/neu.2018.5986

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© Volodymyr Gerzanich et al., 2018; Published by Mary Ann Liebert, Inc.

This Open Access article is distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited.

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FIG. 2. — Glial fibrillary acidic protein (GFAP) immunoreactivity dichotomizes human contusion–traumatic brain injury (TBI) specimens. (A-C) Two sections from the same patient, stained with hematoxylin and eosin (A) or immunolabeled for GFAP (B, C), with the latter shown at low (B) and high (C) magnification: Note absence of specific immunoreactivity for GFAP in the specimen on the left vs. specific labeling of stellate-shaped cells in the companion specimen on the right. (case #2, 11 days post-TBI).