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. Author manuscript; available in PMC: 2019 Apr 3.
Published in final edited form as: Genes Chromosomes Cancer. 2015 May 29;54(8):463–471. doi: 10.1002/gcc.22254

TABLE 2.

Summary of All PRKAR1A Mutations Identified in This Study, Correlation with 17q CN Status and Comparison to PRKAR1A Mutation Database (http://prkar1a.nichd.nih.gov)

SCH # PRKAR1A Exon PRKAR1A cDNA Effect of the mutation Sanger confirmation Reported in PRKAR1A mutation database 17q Status by SNP-array Both Alleles of PRKAR1A Inactivated
SCH1 5 c.441-3C>A Likely alters the splice acceptor site NA No monosomy 17 Yes
SCH2 6 c.535C>T p.Q179X NA No CN-LOH 17q Yes
SCH3 3 c.286C>T p.R96X Confirmed Yes trisomy of 17q21.3-25.3-LOH (UPT) Yes
SCH4 8 c.761_762delCT p.S254Yfs×15 Confirmed No CN-LOH 17q Yes
SCH5 2 c.124C>T p.R42X Confirmed Yes Monosomy 17 Yes
SCH6 11 c.1039T>C p.C347R Confirmed No Monosomy 17 Yes
SCH7 2 c.124C>T p.R42X Confirmed Yes Normal Yes
7 c.708+1G>C exon 7 skipped (shorter PRKAR1A protein) Confirmed Yes
SCH8 5 c.441-1G>C Alters the splice acceptor site Confirmed No Normal Yes
5 c.496C>T p.Q166X Confirmed Yes
SCH9 9 c.851dupT p.Q285Afs×7 NA No Monosomy 17 Yes
SCH10 4 c.380_402del23 p.A127Efs×8 Confirmed No Monosomy 17 Yes
SCH11 10 c.944delT p.V315Gfs×16 Confirmed No Normal No
SCH12 8 c.750_761delinsG p.S251Yfs×15 Confirmed No CN-LOH of 17 Yes

NA: not available, confirmatory Sanger sequencing could not be performed due to sample insufficiency CN-LOH, copy neutral-loss of heterozygosity; UPT, uniparental trisomy.