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. 2019 Apr 4;18:30. doi: 10.1186/s12940-019-0471-9

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Flow chart of this study. The subjects were recruited form the cross-sectional survey on the workers with occupational noise exposure. Then 476 NIHL subjects and matched controls were genotyped. NIHL-associated SNPs were screened and GRS model was constructed. Four hundred eighty-five subjects with non-NIHL at baseline were further followed up and NIHL-associated SNPs and GRS model were further validated