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. Author manuscript; available in PMC: 2020 Apr 1.
Published in final edited form as: Fam Cancer. 2019 Apr;18(2):211–219. doi: 10.1007/s10689-018-00117-1

Table 1:

Prevalence of LS and other cancer pre-disposition genes by multi-gene panel testing

Reference Study population Germline pathogenic mutation carriers
n (% of total cohort)
Prevalence of LS

n (% of total cohort)
Prevalence of non-LS mutations
n (% of total cohort)
VUS rate
n (%)
Colon cancer patients (unselected by age, personal/family history, MSI/MMR-D status)
Yurgelun (2017)21 1058 patients with CRC, clinic-based cohort 105 (9.9%) 33 (3.1%)

MLH1: 13 (1.2%)
MSH2: 7 (0.7%)
MSH6: 6 (0.6%)
PMS2: 7 (0.7%)
Overall: 74 (7%)*

APC: 5 (0.5%); biallelic MUTYH: 3 (0.3%)

BRCA1: 3 (0.3%); BRCA2: 8 (0.8); PALB2: 2 (0.2%); CKDN2A: 1 (0.1%); TP53: 1 (0.1%)
330 (31.2%)
Early-onset colon cancer patients (age < 50 years)
Pearlman (2017)44 450 early-onset CRC patients, population-based cohort 72 (16%) Overall: 37 (8.2%)

MLH1: 13 (2.9%)
MSH2: 17 (3.8%)
MSH6: 2 (0.4%)
PMS2: 5 (1.1%)
Overall: 34 (7.6%)*

APC: 6 (1.3%); biallelic MUTYH: 4 (0.9%), SMAD4: 1 (0.7%)

ATM: 4 (0.9%); BRCA1: 2 (0.4%); BRCA2: 4 (0.9%); CDKN2A: 1 (2%); CHEK2: 1 (0.7%); PALB2: 2 (0.4%)
145 (32.2%)
Stoffel (2018)45 430 early-onset CRC patients, clinic-based cohort 79 (18.4%) 56 (13%)

MLH1: 24 (5.6%)
MSH2: 25 (5.8%)
MSH6: 5 (1.2%)
PMS2: 2 (0.5%)
Overall: 23 (5.3%)

APC: 10 (2.3%); MUTYH: 8 (1.9%)

BRCA1: 1 (0.2%); CHEK2: 1 (0.2%); SMAD4: 2 (0.5%); TP53: 1 (0.2%),
21 (4.9%)
High risk patients (personal history of LS-associated cancer and/or colorectal polyps)
Yurgelun (2015)42 1260 patients referred for LS testing, commercial testing laboratory cohort 182 (14.4%) 114(9.0%)

MLH1: 31 (27%)
MSH2: 40 (35%)
MSH6: 26 (23%)
PMS2: 14 (12%)
EPCAM: 3 (3%)
Overall: 71 (5.6%)*

APC: 5 (0.4%); biallelic MUTYH: 3 (0.2%); STK11: 1 (0.1%) BRCA1: 6 (0.5%), BRCA2: 9 (0.7%)
479 (38%)
Endometrial cancer patients (unselected by age, personal/family history, MSI/MMR status)
Ring (2016)43 381 EC patients, clinic-based cohort 35 (9%) 22 (6%)

MLH1: 3 (0.8%)
MSH2: 5 (1.3%)
MSH6: 6 (1.6%)
PMS2: 6 (1.6%) EPCAM-MSH2: 2 (0.5%)
Overall: 13 (3%)

APC: 1 (0.3%); ATM: 1 (0.3%); BARD1: 1 (0.3%); BRCA1: 1 (0.3%); BRCA2: 1 (0.3%); BRIP1: 1 (0.3%); CHEK2: 4 (1%); NBN: 1 (0.3%); PTEN: 1 (0.3%); RAD51C: 1 (0.3%)
Not reported
*

Includes some moderate and lower penetrant genes not listed in this table

APC, adenomatous polyposis coli; ATM, ataxia-telangiesctasia mutated; BARD1, BRCA1 associated RING domain 1; BRCA1, breast cancer DNA repair-associated gene 1; BRCA2, breast cancer DNA repair-associated gene 2; BRIP1, BRCA1 interacting protein C-terminal helicase 1; CDKN2A, cyclin-dependent kinase Inhibitor 2A; CHEK2, checkpoint kinase 2; CRC, colorectal cancer; EC, endometrial cancer; EPCAM, epithelial cellular adhesion molecule; LS, Lynch syndrome; MLH1, MutL homolog 1; MSH2, MutS homolog 2; MSH6, MutS Homolog 6; PALB2, partner and localizer of BRCA2; PMS2, PMS1 homolog 2; PTEN, phosphatase and tensin homolog; MUTYH, mutY DNA glycosylase; NBN, nibrin; PTEN, phosphatase and tensin homolog; RAD51C, RAD51 paralog C; SMAD4, SMAD family member 4; STK11, serine/threonine kinase 11; VUS, variant of undetermined significance