Kidneys can filter, reabsorb and lose HDL and its components. All these processes are activated when injury involves disruption of the glomerular filtration barrier; renal injury activates hepatic production and metabolism of HDL and its components; renal injury activates intestinal production of apoAI and loads cargo onto HDL modifying the particles.
apoAI, apolipoprotein AI; apoAIV, apolipoprotein IV; HDL, high density lipoprotein; LCAT, lecithin cholesteryl ester acyltransferase; PLTP, phospholipid transfer protein; HL, hepatic lipase; CETP, cholesterol ester transfer protein; SRBI, scavenger receptor BI.