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Proposed mechanisms for rheumatoid arthritis resulting from immune, inflammatory and indirect effects of depression. HPA, hypothalamic-pituitary-adrenal axis; IDO, indoleamine 2,3-dioxygenase; βCaMKII, calcium/calmodulin-dependent protein kinases type II; COX-2, cyclo-oxygenase-2; PGE2, prostaglandin E2.
