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. 2019 Apr 10;19:59. doi: 10.1186/s12883-019-1274-x

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Neuroradiologic imaging pattern of primary brain amyloidoma. The T1-weighted MRI sequence shows a slightly hypointense right temporal mass (arrows in a), an ill-defined, moderate gadolinium enhancement (arrows in b), both compatible with malignant glioma. The CT displays small focal calcifications (arrows in c). The corresponding T2- and FLAIR MRI sequences displayed a hyperintense signal extending beyond the margins of the contrast enhancement indicating peritumoral edema or an infiltrating part of the mass (arrows in d and e). Almost 2 years following surgical removal of the PBA, the T1-weighted MRI sequence shows contrast enhancement at the resection margins (arrows in f) compatible with residual amyloidoma that remained unchanged during the 8-year follow-up