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. 2019 Mar 1;120(7):746–753. doi: 10.1038/s41416-019-0387-8

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Genetic knockdown of CBX2 impedes breast cancer cell growth. The cell growth rate for MCF7 breast cancer cells was calculated over a 7-day period following transfection of anti-CBX2 or scrambled siRNA. Adherent (alive) fractions of cells were counted. Each point represents one cell count from one of three biological replicates, each with two technical replicates. The three growth phases are depicted underneath each plot. KD eff. = CBX2 knockdown efficiency