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. 2019 Apr 10;14:2591–2605. doi: 10.2147/IJN.S195223

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© 2019 Hajiramezanali et al. This work is published and licensed by Dove Medical Press Limited

The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.

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(A) Saturation binding study of ^99mTc–HYNIC–GABA–BN (7–14) to GRP receptors of T-47D cells and (B) inhibition of ^99mTc–HYNIC–GABA–BN (7–14) binding to T-47D cells with the various concentrations of DOTA–BN–TMC–MNPs. Results of a representative experiment are expressed as the percentage of radioactivity bound to the cells (mean±SD, n=3).

Abbreviations: ^99mTc, Technetium-99m; HYNIC, hydrazinonicotinamide; GABA, gamma-aminobutyric acid; BN, bombesin; GRP, gastrin-releasing peptide; DOTA, S-2-(4-isothiocyanatobenzyl)-1,4,7,10-tetraazacyclododecane tetraacetic acid; TMC, N,N,N-trimethyl chitosan; MNP, magnetic nanoparticle.