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. 2019 Apr 15;5:89. doi: 10.1038/s41420-019-0167-5

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Upregulation of FAM129A mRNA levels in ccRCC cancer tissues; normal tissues served as controls. b The upregulation efficiency of FAM129A in tissue samples. c, d Correlations between the FAM129A mRNA expression level and the TNM stage and Fuhrman grades of ccRCC. e Representative images of FAM129A IHC in ccRCC cancer tissues and normal tissues. f, g Western blots of FAM129A showed alterations in protein levels consistent with variations in mRNA levels in clinical samples. h Protein expression of FAM129A is high compared with normal tissues. Correlations between FAM129A protein levels and the TNM stage (i) and Fuhrman grades (j) of ccRCC are shown. ccRCC clear cell renal cell carcinoma, IHC immunohistochemistry. *P values < 0.05, **0.01, ****0.0001