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. 2019 Apr 16;52:22. doi: 10.1186/s40659-019-0231-x

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Schematic representation of a putative mechanism underlying the forming of perivascular resistance niche (PVRN) after exposure to IR. CK2 inhibition suppresses the phosphorylation of NF-κB, then reduces the increase of IL-8 and IL-6 in endothelial cells after exposure to IR, which destroyed the forming of PVRN and finally leads to radiosensitize lung cancer cells (→ indicates stimulation effect, ┫means inhibitory effect)