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. 2019 Mar 15;19(5):3696–3706. doi: 10.3892/mmr.2019.10045

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Copyright: © Tang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — mRNA + lncRNA microarray analysis of kidney allograft tissues. (A) Clustering analysis of the mRNAs revealed that 81 mRNAs were significantly differentially expressed (FC>2.0; P<0.05) between the CAN group (C1-3) and the AMD3100 group (A1-3). mRNA + lncRNA microarray analysis of kidney allograft tissues. (B) Cluster analysis of lncRNAs demonstrated that 140 lncRNAs were significantly differentially expressed (FC>2.0; P<0.05) between the two groups. mRNA + lncRNA microarray analysis of kidney allograft tissues. (C) The top 15 pathways suggested by the Kyoto Encyclopedia of Genes and Genomes pathway analysis. FC, fold change; CAN, chronic allograft nephropathy; lncRNA, long noncoding RNA.

Figure 2. — mRNA + lncRNA microarray analysis of kidney allograft tissues. (A) Clustering analysis of the mRNAs revealed that 81 mRNAs were significantly differentially expressed (FC>2.0; P<0.05) between the CAN group (C1-3) and the AMD3100 group (A1-3). mRNA + lncRNA microarray analysis of kidney allograft tissues. (B) Cluster analysis of lncRNAs demonstrated that 140 lncRNAs were significantly differentially expressed (FC>2.0; P<0.05) between the two groups. mRNA + lncRNA microarray analysis of kidney allograft tissues. (C) The top 15 pathways suggested by the Kyoto Encyclopedia of Genes and Genomes pathway analysis. FC, fold change; CAN, chronic allograft nephropathy; lncRNA, long noncoding RNA.