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. 2019 Apr 18;10(5):338. doi: 10.1038/s41419-019-1570-9

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Genome-wide heatmap analysis indicating the co-occurrence of BRD9-binding sites with active chromatin regions. b Pie chart showing dominant gene silencing observed upon BRD9 KD for genes associated with C2 and C3, suggesting BRD9 as a transcription activator. However, there is no significant difference between numbers of activated and inactivated genes in C2 and C3 clusters. c GSEA analysis showing the top differential hallmark gene sets (nominal P < 0.05) associated with SCR and shBRD9 cells (NES = normalized enrichment score)