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. 2019 Apr 19;9:6316. doi: 10.1038/s41598-019-42838-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fenofibrate did not induce apoptosis in MS1 VEGF angiosarcoma cells. Data were generated in MS1 VEGF angiosarcoma cells by flow cytometry. (a–c) Example experiment showing the proportion (%) of dead, early apoptotic, or dead/late apoptotic cells after treatment with 50 μM fenofibrate (feno) or 1 μM staurosporine (stausp). Staining for FITC-conjugated Annexin V was used as a marker for early apoptosis whereas propidium iodide (PI) staining was used as a marker for cell death. (d,e) Mean data for experiments exemplified in a-c (n = 4, fenofibrate data set; n = 3, staurosporine data set). Data were analysed using a Student’s t-test. *P < 0.05; ****P < 0.0001; n.s, not significant.