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. 2019 Apr 22;7(8):e14047. doi: 10.14814/phy2.14047

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© 2019 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of The Physiological Society and the American Physiological Society

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Nitric oxide dependence of lactation–induced increase in IPSC burst incidence. (A) Representative traces of IPSC bursts recorded in the NOS inhibitor L‐NAME (100 μmol/L) from a virgin rat (upper) and a lactating rat (lower). (B, C) Average incidence of IPSC bursts (burst/h) and percent of MNCs displaying IPSC bursts (% cells_burst) recorded in the presence of 100‐μmol/L L‐NAME in slices from virgin and lactating rats. NOS inhibition abolished the increase in IPSC burst incidence in OT MNCs and the increased percentage of OT MNCs that generated IPSC bursts in slices from lactating rats.